Search USGS⌕ Search

Geology topics

Glenn H. Olsen

Publications and source records attributed to Glenn H. Olsen.

At least 91 records · Page 5Linked to original sources
USGS2000

Limb dysfunction

Explore the source record for details and available documents.

Book chapter↗
USGS1997

Efficacy of eastern encephalitis immunization in whooping cranes

An epizootic of eastern equine encephalitis (EEE) at the Patuxent Wildlife Research Center (PWRC), Laurel, Maryland (USA), in 1989 provided an opportunity to determine if EEE immunization protected whooping cranes ( Grus americana ). Based on seroconversion of 31% of sympatric hatch-year sandhill cranes, Grus canadensis , and a previous 35% case fatality rate in whooping cranes, 17 (37%) of the 46 susceptible whooping cranes should have been exposed to virus and six should have died. As there were no deaths in these birds, the EEE vaccination program appeared to be efficacious in this whooping crane population.

Journal of Wildlife Diseases↗
USGS1997

Whooping crane mortality at Patuxent Wildlife Research Center, 1982-95

Whooping cranes (Grus americana) have been reared at Patuxent Wildlife Research Center since 1966. During 1982-95 there were 103 mortalities caused by infectious and parasitic diseases (46%), trauma (21%), anatomic abnormalities (17%), miscellaneous conditions (12%), and open or no diagnoses (5%). The implications that disease may have on new whooping crane flocks in Florida and Canada are discussed, based on these mortality factors in captivity.

Book chapter↗
USGS1997

Causes of Mississippi sandhill crane mortality in captivity 1984-95

During 1984-95, 111 deaths were documented in the captive flock of Mississippi sandhill cranes (Grus canadensis pulla) housed at the Paluxent Wildlife Research Center. Trauma was the leading cause of death (37%), followed by infectious/parasitic diseases (25%), anatomic abnormalities (15%), and miscellaneous (8%). No positive diagnosis of cause of death was found in 19% of the necropsies. Chicks < 2 months old suffered 76% of captive deaths. Trauma, the greatest cause of deaths of captive juveniles anti adults, is likely limited to collisions in the wild. lnfectious/parasitic diseases and anatomic abnormalities could affect wild chick survival at similar rates to those of captive chicks.

Book chapter↗
USGS1997

Cranes

Explore the source record for details and available documents.

Book chapter↗
USGS1995

Accumulation and loss of arsenic and boron, alone and in combination, in mallard ducks

Arsenic and boron are common in the environment, and wildlife can be exposed to toxic concentrations through both natural and human-influenced processes. We exposed adult male mallard ducks to dietary concentrations of 300 ppm arsenic as sodium arsenate, 1,600 ppm boron as boric acid, or both and estimated the tissue accumulation and loss rates when the ducks were returned to uncontaminated food. Both elements were accumulated rapidly; equilibrium levels were reached for arsenic in 10 to 30 d and for boron in 2 to 15 d. Accumulation of each element was slowed by the presence of the other in the diet. Boron was eliminated by mallards very rapidly, with few detectable residues ≥ 1 d after removal of boron from the diet; arsenic was also rapidly lost with half-lives of 1 to 3 d (half-lives were not constant throughout the loss period). Arsenic loss rate was not affected by the presence of boron. Arsenic accumulated to the highest level in liver tissue, with blood and brain levels lower; concentrations in the liver and blood were proportional but affected by the presence of boron. Boron concentrations were highest in the blood, followed by the brain and liver; concentrations in the blood and liver were proportional.

Environmental Toxicology and Chemistry↗