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C. E. Smith

Publications and source records attributed to C. E. Smith.

10 recordsLinked to original sources

Velocities of the Smith Glacier Ice Tongue and Dotson Ice Shelf, Walgreen Coast, Marie Byrd Land, West Antarctica

Velocity measurements were made for two time intervals on the Smith Glacier ice tongue (1973–88 and 1988–90) and three on the Dotson Ice Shelf (1972–88, 1973–88 and 1988–90). The Smith Glacier ice tongue velocities for the two intervals are similar near the grounding line but show a progressive increase toward the terminus in the late 1980s. The Dotson Ice Shelf velocities remained virtually constant during all three time intervals. The increased velocities of the Smith Glacier ice tongue may be attributed to a general loss of densely packed icebergs that buttressed the terminus during the 1970s but drifted out to sea during the late 1980s. The Smith Glacier ice tongue receded as much as 10 km between 1973 and 1988, the Dotson lee Shelf 5–7 km in the same time period. Similar observations of drifting and ca1ving were made for the adjacent Thwaites Glacier ice tongue. The cause of the loss of ice in the region is unknown but it may have been a change in winds or a warming of the air or water during the late 1980s.

Annals of Glaciology

Herpesvirus salmonis: pathological changes in parenterally-infected rainbow trout, Salmo gairdneri Richardson, fry

After fry of rainbow trout, Salmo gairdneri Richardson, had been infected parenterally with Herpesvirus salmonis , moribund or freshly dead specimens were examined histopathologically. The virus produced a generalized infection, the first signs of which appeared after 2–3 weeks. Visceral and respiratory organs and the heart showed major pathological changes, and pancreatic syncytia were judged to be pathognomonic. Kidneys were prime targets for the virus and showed the highest levels of infectivity; lesser amounts of virus were present in the stomach, liver, and intestine. The virus did not spread by contact to produce clinical disease nor could disease or viral replication be induced by parenteral inoculation of yearling Oncorhynchus nerka (Walbaum).

Journal of Fish Diseases

A lymphosarcoma of thymic origin in the rainbow trout, Salmo gairdneri Richardson

A rainbow trout with a malignant lymphoma of probable thymic origin showed direct spread of the tumour to the gills, metastases to the liver and head kidney, and the presence of abnormal circulating lymphocytes. The cells of the tumour possessed plasma membrane immunoglobulin detectable by immuno–fluorescence. Examination of the tumour cells by transmission electron microscopy revealed no evidence of virus associated with the cells. Infectious viruses could not be detected in the tumour tissue, and the tumour was not transmissible by injection of live tumour cells into young rainbow trout. Attempts to establish the tumour in long–term culture were unsuccessful.

Journal of Fish Diseases

Characterization of the host response to the myxosporean parasite, Ceratomyxa shasta (Noble), by histology, scanning electron microscopy, and immunological techniques

The tissue response of Salmo gairdneri Richardson, against the myxosporean parasite. Ceratomyxa shasta (Noble), was investigated using histological techniques, scanning electron microscopy and immunological methods. The progress of infection in C. shasta ‐susceptible and resistant steelhead and rainbow trout was examined by standard histological techniques and by indirect fluorescent antibody methods using monoclonal antibodies directed against C. shasta antigens. Trophozoite stages were first observed in the posterior intestine and there was indication that resistance was due to the inability of the parasite to penetrate this tissue rather than to an inflammatory response. Examination of a severely infected intestine by scanning electron microscopy showed extensive destruction of the mucosal folds of the posterior intestine. Western blotting and indirect fluorescent antibody techniques were used to investigate the immunological component of the host response. No antibodies specific for C. shasta were detected by either method.

Journal of Fish Diseases

Accumulation, sublethal effects, and safe concentration of a refined oil as evaluated with cutthroat trout

Cutthroat trout ( Salmo clarki ) were exposed for 90 days in the laboratory to a refined oil collected from the North Platte River at a seepage site below the American Oil Company refinery at Casper, Wyoming. Fish were exposed to five concentrations and a control, and seven biological responses (survival, growth, gill pathology, liver pathology, caudal fin erosion, caudal fin pathology and swimming performance) were correlated with water concentration and tissue accumulation of petroleum hydrocarbons. Fish in the highest water concentration, 183 μ g/L total oil, accumulated tissue concentrations of 4.6 μ g/g total naphthalenes and responded adversely to all seven biological measurements. Cutthroat trout in the lowest water concentration, 24 μ g/L total oil, had a mean tissue concentration of 1.2 μ g/g total naphthalenes, but their response was similar to that of the control fish to all seven biological responses. Fish exposed to 39 μ g/L water concentration accumulated tissue concentrations of 2.7 μ g/g and responded negatively to 2 of the 7 biological measurements. Therefore, the maximum safe limit for this oil and cutthroat trout is between 24 and 39 μ g/L.

Archives of Environmental Contamination and Toxico

Comparative susceptibility of Atlantic salmon (Salmo salar) to the enteric redmouth bacterium and Aeromonas salmonicida

The bacterium causing enteric redmouth (ERM) and Aeromonas salmonicida were found to be equally pathogenic for fingerling Atlantic salmon ( Salmo salar ). Injection of 5 × 10 5 cells of ERM or A. salmonicida killed all salmon within 96 h. After a 30 min exposure to water-borne cells of the two test bacteria about one-half of the test salmon died within 14 days. Both ERM and A. salmonicida were transmitted horizontally. Results indicate that efforts should be made to prevent introduction of ERM into watersheds where Atlantic salmon occur.

Journal of Wildlife Diseases